Sentence examples for model of human breast from inspiring English sources

Thibaudeau, L. et al. A tissue-engineered humanized xenograft model of human breast cancer metastasis to bone.

White, D. E. et al. Targeted disruption of beta1-integrin in a transgenic mouse model of human breast cancer reveals an essential role in mammary tumor induction.

Osborne, C. K. et al. Comparison of the effects of a pure steroidal antiestrogen with those of tamoxifen in a model of human breast cancer.

We then transduced the iPS-derived MSCs to express cytosine deaminase and injected them locally into a mouse xenogeneic model of human breast cancer.

These multiple roles of miR-34a are maintained in a model of human breast cancer, in which chronic expression of miR-34a in triple-negative mesenchymal-like cells (enriched in cancer stem cells CSCs) could promote a luminal-like differentiation programme, restrict the CSC pool, and inhibit tumour propagation.

Delivery of pRL-CMV from a SELP hydrogel resulted in increased transfection in a murine model of human breast cancer by 1 3 orders of magnitude, as compared to naked DNA.

Using a xenograft mouse model of human breast cancer, fluorescent imaging showed that only HE-MAP could effectively target GST to the tumor site, while reducing non-specific association of MAP in other organs.

Significantly, FCW81 displays therapeutic efficacy in a xenograft model of human breast cancer by not only serving to inhibit markedly the growth of cancer cells but also to block effectively cancer cell metastasis.

Figure 4 VLP H1 and VLP H2 suppressed tumor growth in a xenograft model of human breast cancer.

In a mouse model of human breast cancer, fibroblast growth factor receptor 1-induced mammary tumorigenesis is associated with local production of IL-1β and can be blocked by treatment with an IL-1β neutralizing antibody (Reed et al., 2009).

Kluger HM, Lev DC, Kluger Y, McCarthy MM, Kiriakova G, Camp RL, Rimm DL, Price JE. (2005) Using a Xenograft Model of Human Breast Cancer Metastasis to Find Genes Associated with Clinically Aggressive Disease.