Your English writing platform
Discover LudwigExact(60)
For years, they were wedded to the wrong model of heart disease.
Calcineurin-induced energy wasting in a transgenic mouse model of heart failure.
In addition, the method is demonstrated through application to a model of heart disease in diabetes.
But the new model of heart disease shows that the vast majority of heart attacks do not originate with obstructions that narrow arteries.
Kawase, Y. et al. Reversal of cardiac dysfunction after long-term expression of SERCA2a by gene transfer in a pre-clinical model of heart failure.
Byrne, M. J. et al. Recirculating cardiac delivery of AAV2/1SERCA2a improves myocardial function in an experimental model of heart failure in large animals.
Kawase, Y. et al. Reversal of cardiac dysfunction after long-yerm expression of SERCA2a by gene transfer in a pre-clinical model of heart failure.
del Monte, F. et al. Improvement in survival and cardiac metabolism after gene transfer of sarcoplasmic reticulum Ca2+-ATPase in a rat model of heart failure.
del Monte, F. et al. Improvement in survival and cardiac metabolism after gene transfer of sarcoplasmic reticulum Ca(2+ -ATPase in a rat model of heart failure.
In this study, we use a multiscale approach to analyze a model of heart failure and connect its results to features of the electrocardiogram (ECG).
Consistent with our hypothesis that TNF constitutively drives myogenic signalling in skeletal muscle resistance arteries, ETN abolishes myogenic responsiveness in an experimental mouse model of heart failure (HF; Fig. 1a,b), rather than simply normalizing it.
Write better and faster with AI suggestions while staying true to your unique style.
Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com