Sentence examples for model of graft from inspiring English sources

We investigated the efficacy of galectin-1 in a murine model of graft versus host disease and found that 68% of galectin-1-treated mice survived, compared to 3% of vehicle-treated mice.

Moreover, in a mouse model of graft versus host disease (GvHD), injection of allogeneic MSCs had no beneficial effect on the GvHD [ 56].

Another study utilizing a mouse model of graft versus host disease (GVHD) also linked diminished Paneth cell antimicrobials to an increase of normally rare septicemia causing E. coli in expense of symbiotic diversity (Eriguchi et al, 2012).

Also, the protective effects of SAHA in a murine model of graft versus host disease are associated with a block in the rapid accumulation of phosphorylated STAT1 in the liver and the spleen [ 52].

Furthermore, this mutein prolonged survival in a model of graft-versus-host disease and blocked spontaneous proliferation of smoldering adult T cell leukemia (ATL) T cells.

We used a humanized, lymphocyte transfer model of graft-versus-host disease, to evaluate the durability of protein transduction domain mimic Anti-pPKCθ mimic Anti-pPKCθ delivered into humodulationeral mononuclear blood cells ex vivo.

Highfill et al. reported that the adoptive transfer of MDSCs significantly improved survival in a model of graft-versus-host disease [ 67].

For example, in a rodent model of graft-vs.-host disease (GVHD) vorinostat reduced inflammation and GVHD-related mortality while Wang et al. found that panobinostat induced a Th1-directed pro-inflammatory response and augmented GVHD progression.

For example, IL-22, expressed by Th cells and ILCs and with a receptor located on stromal mesenchymal and epithelial cells, triggers the production of genes involved in epithelial cell differentiation and survival and has been shown to protect intestinal Paneth and stem cells against immune-mediated tissue damage in a model of graft-versus-host disease in mice.

With regard to anatomic reconstruction techniques, evidence from animal models of graft-bone healing show that the four tissue zones characteristic of the fibrocartilaginous enthesis are not regenerated (Lu and Thomopoulos 2013).

For the experimental model of skin graft, the proceeding described by Billingham and Medawar was employed [46].