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The genetics of different flower architectures can be explained by the ABC model of floral organ identity and its variants.
The premise for these mutations is explained in the classic "ABC model" of floral development for Arabidopsis [ 1, 2].
According to the canonical ABC model of floral development (Weigel and Meyerowitz 1994), differential gene expression results in the specification of the identity of the various floral organs.
For instance, floral organ identity genes (homeotic genes) of the ABCDE model of floral organ development are mostly MADS genes [ 44].
They act in a combinatorial manner as predicted by the well-supported (A BCE model of floral organ identity specification [ 50– 50].
Of these target genes, 62 belonged to MADS-box genes in the expanded ABCDE model of floral development, which determines identity of flower organs [ 28].
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In the past, the approach has been successfully applied for diverse systems such as gene regulatory networks (Albert and Othmer, 2003; Chaves et al., 2005), models of floral morphogenesis (Mendoza et al., 1999), mammalian cell cycle (Mendoza, 2006), EGFR signalling (Samaga et al., 2009) or apoptosis (Schlatter et al., 2009).
Related sites All about enantiostyly (with nice pictures) Arabidopsis model system of floral development.
A network is formally defined and derived for seed dispersion model of target floral species where vertices represent habitat patches which are connected by an edge if the distance between the patches is less than a threshold distance.
All MADS genes in the ABCDE model of plant floral development are MIKC-type MADS.
Based on the relative accumulation of kiwifruit MADS-box gene transcripts in the wild-type male and female flowers and 'Pukekohe dwarf' mutant flowers, a model of kiwifruit floral organ specification was proposed.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com