Sentence examples for model of coronary from inspiring English sources
An open-chest model of coronary occlusion and acute ischemia was used in 14 adult pigs.
We previously demonstrated that adenoviral-mediated CRNK gene transfer improved survival and LV function, and slowed LV remodeling in a rat model of coronary artery ligation-induced HF.
This study was designed to determine in a dog model of coronary thrombosis whether short-term eptifibatide (Ep) combined with low-dose plasminogen activator (rt-PA) inhibits platelet recruitment at sites of endothelial damage after normalization of platelet function.
Thus, the Paigen diet-fed PDZK1/apoE dKO mice represent a new murine model of coronary heart disease and suggest that PDZK1 may represent a valuable target for therapeutic intervention.
Present study observed the pathological and angiographic evolution in a pig model of coronary slow flow.
Injection of human CRP into a rat model of coronary artery disease reproducibly enhanced infarct size by ~40% (40).
Interestingly, PCr treatment reduced necrotic tissue injury and improved contractile function in animal models of coronary artery ligation [102] and ischemia reperfusion injury [103].
Its vascular effects were evaluated in two models of coronary vascular reactivity in primates: 1) the paradoxical vasoconstriction to acethylcholine (Ach) coronary infusion after 5 months of mildly atherogenic diet in ovariectomized (OVX) Cynomolgus monkeys and 2) the pharmacologically evoked coronary vasospasm in the OVX Rhesus monkey.
The flow fields inside three polydimethylsiloxane (PDMS) models of coronary artery bypass grafts, including the coupled SQA graft design, a conventional ETS anastomosis, and a parallel side-to-side (STS) anastomosis, are investigated under pulsatile flow conditions using particle image velocimetry (PIV).
For example non-transgenic models of coronary artery disease involve acute ischemia rather than progressive atherosclerotic coronary disease which would trigger cardiac remodeling [ 1].
Some researches on combinations of several growth factors have been explored in animal models of coronary artery disease and peripheral arterial disease.
