Sentence examples for model of cap from inspiring English sources

Lumped model of CAP code uses 2-phase, 3-field equations for fluid behaviors, and has appropriate constitutive equations, 1-dimensional heat conductor model, component models, trip and control models, and special process models.

In the TRAMP mice model of CaP progression, our gene signature alone as determined by RT-qPCR, besides discriminating CaP from benign tissue, also predicted individual response to treatment with Green Tea Catechins (GTCs) [19].

Subsequently, discriminatory analysis and ROC curves were used to establish and assess a predictive diagnostic model of CAP with these two independent factors.

Given that expression of activated Src is sufficient to induce CR-CaP in a tissue recombination model of CaP [ 24], our results suggest that continued Src activation promotes CR-CaP in the CWR22 model.

In our experimental model of CaP growth in the bone, administration of the anti-huRANKL MAb did not significantly inhibit osteolysis associated with the growth of C4-2 cells in the bone, suggesting that the RANKL expressed by these tumor cells is not the driving force for tumor-associated osteolysis in this model.

The study incorporates methodological innovation in terms of the modelling of CAP budgetary mechanisms.

It has been reported that BMP-6 expression positively correlates with CaP progression [43], [44] and that BMP-6 is the BMP primarily responsible for inducing an osteoblast response in mouse models of CaP bone metastasis [17], [45].

Subsequently, discriminatory analysis and ROC curves were used to establish and assess predictive diagnostic models of CAP by using these four independent factors.

Although it is well known that combined inactivation of PTEN and Tp53 in compound models of CaP greatly accelerates tumor development (Jeet et al., 2010), in terms of disease progression the importance of whether p53 or PTEN loss of function occurs first remains to be determined.

Indeed, DEX has been shown to induce resistance to several cytotoxic agents, including the taxane, paclitaxel, in cells isolated from surgical resections of prostate tumours (Zhang et al, 2006) and to a range of cytotoxics in xenograft models of CaP (Zhang et al, 2007).

To better understand this process, we have studied the effects of different statins in our in-house, well-categorised models of CaP behaviour, evaluating the differential effects of specific statins on cellular binding, migration and early cellular survival in human BMS.