To analyse the changes in miRNA expression profile during epithelial wound repair, we used a previously well established in vitro model mimicking this process [ 23, 32- 34], that allowed real-time monitoring of the rate of epithelial repair and quantitative analysis using time-lapse microscopy.
This model mimics the steps sequential bone morphogenesis and has permitted the isolation of bone morphogens, such as bone morphogenetic proteins (BMPs).
This model mimics the stratified structure of the liver in vivo and incorporates the two major cell types found in this organ.
This model mimics the early pathogenic process of PD where cell death is preceded by reduced neurite outgrowth and synaptic alterations.
This model mimics the morbidity and mortality of immunocompromised hosts such as patients with leukemia, severe burn wounds or recipients of organ transplants [8].
This model mimics the cumulative effect of the reprocessing protocol on flexible endoscopes.
This model mimics the genetic haploinsufficiency of CdLS patients with mutation in NIPBL.
Importantly, this model mimics the dissemination process upon invasive aspergillosis (IA) only to a limited extent.
53 This model mimics the syndromic human disorder, Ichthyosis Follicularis, Atrichia and Photophobia (IFAP) syndrome (OMIM #308205).
Second, this model mimics the trophoblast functions more realistically, including production and release of secretory components, proliferation, growth, and differentiation.
