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b Cross-sectional topographic profiles of the basin, extracted from the MOLA topography model in panel a. c Geology map of the study area.

Furthermore, referring to the final model in panel A, several of our control variables (MV, IFF as well as the index dummies) are significant and show the predicted signs, which indicates that characteristics of a firm's information environment are relevant for the width of bid ask spreads.

Indeed, our model reveals that system optimization of individual myelinated axons is achieved when the inner diameter approaches 77% of the outer diameter, i.e. a g-ratio of 0.76 0.77 (Figure 3A D; theoretical model in panel B: r = 1.0, R2 = 0.99, p<0.0001; model fit to experimental data in panel D: R2>0.96 for each).

The inverse nature of these associations by genotype, from the blood lead model in panel 2 of Table 3, is shown graphically in Figure 1.

We felt that the old model in panel A was needed to contrast with the new model in panel B. We also felt that panels C and D were needed to explain the changes that occur with LTP (panel C) and LTD (panel D).

Structures in panels A – C are aligned with the no-nucleotide K349 model in panel D by strand b7 of the central beta sheet, which connects to the switch II loop; this alignment approximately maintains the orientation of the upper subdomain, thus preserving a contact between L8 within this subdomain and alpha tubulin (see Figure 2 ).

Solid lines in a are the fittings of the experimental data using the model in panels b and c.

Models in Panel a and Panel b are otherwise identical; other controls are shown at the bottom of the table.

The relative head domain orientations of the II-c and II-e subclass density maps are indicated as cartoon models in panel (E) and (G), respectively.

Models in Panel A include separate controls for short and long term bank debt as a share of the firms turn over, the log of the firm's turn over, the log of workers and indicators for zero values of sales and debt.

Figure 3 figure supplement 1 depicts a peroxisome biogenesis model, referred to as Model 2, alternative to the model depicted in panel (A ). Figure 3 figure supplement 2 depicts data similar to panels (B – D ) but with Pex3-mRFP as the peroxisome marker.