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Plasma TC was also increased by 16.30% in the model group but the difference was not statistically significant (P = 0.51).
Preterm children were not included in the Model group, but predictions for this group were within the expected range.
The HDL-C and LDL-C levels in the control group were lower than those in the model group, but not significantly (P > 0.05; Figure 1).
The rats in the Sham group were subjected to the same procedures as the rats in the Model group but the MCA origin was not occluded.
In UDCA and dexamethasone group the rate was also significantly lower than that in model group but higher than that in corilagin group (P<0.05).
The L.M1-16 group had more adipocytes under each field of view than the model group, but the number was less than the control and L.9-41-A groups (p < 0.05).
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They were all slightly higher than their model groups, but it had no statistical significance.
From week 8 to week 12, it was obvious that the histological analysis identified necrotic bone cells surrounded by necrotic bone marrow and necrotic regions with or without being repaired near the epiphyses and subchondral bone in the model groups, but slightly in the treatment groups.
The level of Aβ1-40 in Timosaponin-BII treated group was lower than those in model group and vehicle group but remained higher than those in the normal control group.
As shown in Figure 1A and B, the same tendency of ACT and PWT was observed in the model group and fentanyl groups, but ACT remained at a relatively steady level, whereas PWT gradually increased towards baseline levels.
The level of MDA in the experimental group was lower than those in model group and vehicle control group but remained higher than those in the normal control group.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com