Sentence examples for model group both from inspiring English sources

IL-1β significantly increased activities of caspase-8 and caspase-3 in the model group (both P < 0.01; n = 3).

However, the neurological deficit scores were markedly lower in the EA and vehicle + EA groups than in the model group (both P < 0.05; Figure  2B).

IL-1β significantly increased the gene expression of iNOS and NO synthesis in the model group (both P < 0.01; n = 3).

The numbers of BDNF- and pRaf-1-positive cells in the ischemic cortex were significantly higher in the U0126 + EA group than in the model group (both P < 0.05; Figures  3B and 4A; Table  1).

After 7 days of reperfusion, the neurological deficit scores of the EA-5Hz and EA-25Hz groups were significantly lower than those of the Model group (both P < 0.05; Fig.  2b).

However, treatment with ES 30 and 90 mg/kg significantly decreased the NAGL levels compared with the diabetic model group (both P < 0.05), and the level was lower than that observed in AG-treated rats (Fig.  5).

The lungs in the model group and both groups that received saline appeared grossly swollen with diffuse haemorrhage in the lung tissue and pink exudates from the airway.

For the intervention group, this defect was improved from the model group in both T1 (P = 0.004) and T2 (P = 0.014).

TNF-α concentration in the BALF of the rats in the PPS-aer and PPS-inst groups was significantly reduced when compared with the model group and both groups that received saline (P < 0.01, Figure 4c).

As shown in Figure 4, a significantly decreased lung index (lung/body weight ratio; Figure 4a) and total protein content in BALF were observed in the PPS-aer and PPS-inst groups when compared with the model group and both groups that received saline (P < 0.01).

The results showed that the B-ALL markers expressed highly in the BM cells of model group, while reduced significantly in both rapamycin preventative and treatment groups, further suggesting that activation of autophagy in both prevention and treatment with rapamycin slows down or inhibits B-ALL leukemogenesis.