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Also, a gas/solid reaction model for sulfur dioxide (SO2) removal by lime has been developed.
Using this information, a preliminary model for sulfur assimilation in A. ferrooxidans is presented and possible mechanisms for its biochemical and genetic regulation are discussed.
Figure 1 presents a preliminary model for sulfur uptake and assimilation in A. ferrooxidans derived from a bioinformatic analysis of the potential genes present in its genome.
Using this information, we were able to develop a model for sulfur assimilation in X. citri evidencing the expression of genes of the sulfate uptake and sulfur assimilation pathway.
We posit that the preliminary metabolic model for sulfur uptake and assimilation in A. ferrooxidans presented here has permitted the capture of potentially biologically valid relations that have now been highlighted for experimental validation.
A model for sulfur deprivation responsive gene regulation among the three regulatory proteins SAC1, SNRK2.1 and SAC3 (same as SNRK2.2) has been proposed: SNRK2.2 inhibits SNRK2.1-activated expression of sulfur-responsive genes, and the active SAC1 unblocks the inhibition of SNRK2.1 by SNRK2.2 in sulfur-deficient conditions for full expression of sulfur-responsive genes [ 25, 81].
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Chemical analysis of the superatom model for sulfur-stabilized gold nanoparticles.
Synthetic mixtures of dibenzothiophene (DBT) and 4,6-dimethyldibenzothiophene (4,6-DMDBT) in n-hexadecane (n-HD) were used as models for sulfur contaminated diesel fuel.
A rotational isomeric state (RIS) model for polymeric sulfur chains has been used to calculate the concentrations of cyclics Sx with x > 8 in liquid sulfur below the critical polymerization temperature.
This delay was predicted by our model for S > 0 μM, due to slower sulfur decay causing an extended period of Calvin cycle activity.
Gene expression values (log2 ratio of median) for all genes/operons alluded in the revised model of sulfur oxidation in A. ferrooxidans ATCC 23270.
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