Sentence examples for model for spinal from inspiring English sources

The molecular regulation and cellular changes found in salmon vertebral fusions are similar to those found in mammalian deformities, showing that salmon is suitable for studying general bone development and to be a comparative model for spinal deformities.

In an experimental model for spinal cord compression, exposure of juxtaparanodal Kv1.2 channels with accompanying myelin retraction at the nodes contributed to the induction of conduction block (Ouyang et al., 2010).

The fusion process involves molecular regulation and cellular changes similar to those found in mammalian deformities, indicating that salmon is suitable for studying general bone development and to be a comparative model for spinal deformities.

Conversely to these theoretical extrapolations the same author reports further experimental data [ 45] which show contradictory results, in particular reliability of the LQ model for spinal cord as late reacting tissue with a fractionation dose up to 10 Gy.

In humans and in a mouse model for spinal cord lesion, upregulation of FAS ligand (FASLG) is required for the recruitment of peripheral myeloid cells to the injured tissue [ 31].

Calpain activation has been demonstrated to cleave myelin proteins including myelin basic protein and myelin-associated glycoprotein [51] and contribute to myelin disruption in animal models for spinal cord injury [52] and MS [53].

More specifically, severe thoracic lesions result in permanent paralysis of hind limbs in rodents, which have been extensively used as models for spinal cord injury in humans.

However, there is no practical and clinically relevant mouse model available for spinal cord ischemia.

*The Mean (SD) is presented for each variable in the model for each spinal manipulation dose level (0, 6, 12, and 18 manipulation visits) and the total sample.

From the results of our protein domain analysis it is evident that AS plays a major role in disease implications in both human and cow, and is suitable as a model for investigating spinal muscular atrophy, colon cancer, tangier disease, glaucoma, spinocerebellar ataxia, polycystic kidney disease, autoimmune poly grandular syndrome and wilson's disease.

This study was designed to assess the neuroprotective effect of xenon-induced delayed postconditioning on spinal cord ischaemia reperfusion injury (IRI) and to determine the time of administration for best neuroprotection in a rat model of spinal cord IRI.