Sentence examples for model deficiency in from inspiring English sources

Exact(2)

Furthermore, in this same model, deficiency in p21WAF1(Wnt1/p21WAF1-/- Wnt1/p21WAF1-/-1+/- mice) wandunable to mimic the acceleration due to loss of p53 [ 48].

E- selectin knockout mice have also been generated and have defects in neutrophil infiltration in different tissues that are exacerbated when combined with P- selectin knockout mice [ 51], but in an EAE model deficiency in E- selectin does not affect the progression of the disease [ 50].

Similar(58)

In this model, the deficiency in lift arising from the circulatory effect of shed vorticity is modeled using the indicial response function.

Large differences were found in both lumped and distributed modeling cases between the updated and the a priori lower zone tension and primary free water contents for both WC and SC approaches, indicating possible model structural deficiency in describing low flows or evapotranspiration processes for the catchments studied.

We demonstrate using passive cutaneous anaphylaxis, a mast cell-dependent model, that deficiency in ST2 or antibody blockage of ST2 or IL-33 ablated the late phase inflammatory response but that the immediate phase response was unaffected.

In the NOD mouse model, a deficiency in the clearance of apoptotic cells by macrophages has been described [ 69].

Since the analysis in the Rasch model indicated deficiencies in discrimination and model fit, a graded response model was applied, adding parameters for differences in item weights (2PL GRM) [ 23].

Some studies [53, 54] have also highlighted another important problem of the common decision tree-based F0 modeling: its deficiency in capturing the effect of contextual features that are poorly represented in the training database.

Several mouse models with deficiency in the ING1 locus have been described [ 19, 22].

Two zebrafish (Danio rerio) models with deficiency in nipbl 47 and esco2 48 have been generated to explore the molecular mechanisms causing the phenotypes observed in CdLS and Roberts syndrome.

Recently, a detailed analysis of HSC in an AKT1/2 double-deficient mouse model revealed a deficiency in competitive long-term reconstitution (Juntilla et al., 2010).

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