Our model of supply is much richer as it can accommodate any set of lots along an axis – yet for trips from one or two origins only and with no route choice, while network simulation models cover multiple origins and routes.
Given that the average model covers interactions from multiple signaling pathways and is more complex than the pathway diagrams presented in most review papers, even the qualitative analysis of the model is highly challenging.
For real life application areas such as medicine or biotechnology there is a need to scale up and build more complex multiscale models which cover multiple spatial and/or temporal scales [ 6, 7]; the Virtual Physiological Human [ 8] and High-Definition Physiology [ 9] projects are international initiatives attempting to (partially) address this challenge.
A key strength of the toolkit formed by CellNOptR, CNORdt, CNORode and CNORfuzzy is that it covers multiple logic modeling formalisms (Boolean steady-state, Boolean multiple steady-state, Boolean time courses through synchronous update, steady-state constrained fuzzy logic and continuous logic-based ODEs).
This model covers a spatial multiplexing/multiple-access CDMA system using a different spreading code per transmit antenna [2], and is the same as the PARAFAC-CDMA model proposed in the seminal paper [20].
Still considering F=1, in this case we have R=M t, Ω = I M t, and Θ = 1 R T, which implies G = H ̄, and model (21) becomes: Y ̄ 1 = ( C ◇ H ̄ ) S T ∈ ℂ P M r × N. (27) This model covers a spatial multiplexing/multiple-access CDMA system using a different spreading code per transmit antenna [2], and is the same as the PARAFAC-CDMA model proposed in the seminal paper [20].
Harper's Bazaar had zero model covers.
The paper also covers multiple time-steps, holonomic constraints and scalability.
The Bay Area covers multiple municipalities with multiple school districts.
Such models need to cover multiple scales, from molecular pathways in cell-autonomous immunity and intercellular communication pathways in tissue inflammation to whole organism response pathways in systemic disease.
