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In an in vitro rat bladder strip model, contraction inhibition was not observed in case of 125I-BoNT-A.
In the first part of the paper we discuss a multi-dimensional analogue of the well-known construction by D. Clark that allows one to study families of spectral measures of perturbations of the model contraction.
In the LCMV infection model, contraction of CD8+ T cell response was shown to be reduced, but not absent in Bim-deficient mice [7].
In order to accurately model contraction of the cell, we manually change the position and size of each clump's particles to match the amount of contraction predicted by the single-cell model in Section 2.1.
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And, as always, when a space becomes overrun by me-too companies and models, contraction seems inevitable — if not necessary.
Our results indicate that micro-aspiration can be used to test simple mechanical models of contraction.
By formalizing impedance control within the theory of stochastic OFC, we overcome the fundamental inability of energy based optimization methods to model co-contraction.
Our results here – using a larger data set and a model of contractions that takes into account viscoelasticity – suggest that force generation increased with increasing stiffness among clutches: softer clutches appeared to produce lower apical tensions during contractions.
Notably, in our model, co-contraction is achieved without changing the standard energy based cost function since the uncertainty information is contained in the learned internal dynamics function as a stochastic term.
In these models, hydrogel contraction is a popular measure of cell activation.
The Family-Lineage models suggest Contraction on the coelomate lineage and re-expansion on the branch leading to vertebrates.
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