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Transport modeling was based on a one-dimensional diffusion model, assuming thermodynamic equilibrium.
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Despite this universally accepted fact, gene regulation is typically formalized into models that assume thermodynamic equilibrium.
model, assuming stochastic opening and closing.
A model assuming phenotypic assortment fitted the data better than a model assuming social homogamy.
A general consumer-resource model assuming discrete consumers and a continuously structured resource is examined.
We fit the model assuming that the trials are independent.
The thermodynamic model assumes there are PDI disulfide bonds that have electron affinities above and below the nascent proteins disulfide bonds.
This assumption is justified because the thermodynamic model assumes a mathematical expression by decomposition, whose parameterization must be done against experimental data.
While this provides a satisfying thermodynamic explanation, the model assumes that a protein can freely diffuse within the cylindrical confinement of the BTA fiber.
The model assumes an idealised surface topography.
The former model assumed uniform bed material.
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