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Glycemic status is assessed by insulin resistance using HOMA (Homeostasis Model Assessment) and 2-hour plasma glucose level after a standard glucose load.
Part II: Model assessment and application.
The results of model assessment and two case studies were reported.
The model assessment and procedure for determination of material parameters are described.
Taken together, this results in reliable a priori methods for model assessment and calibration.
Finally, we discuss efforts towards establishing unified criteria for map and model assessment and validation.
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Insulin resistance assessed by the homeostasis model assessment index and urinary albumin excretion decreased significantly in the miglitol group but not in the mitiglinide group.
IR by homeostatic model assessment (HOMA2), and β-cell secretion, as measured by insulin, split and intact proinsulin, and C-peptide levels were determined for both groups.
To account for possible H1N1 effects on pancreatic β-cell function, a homeostasis model assessment (Homa) and IAI were used to measure insulin resistance and sensitivity [ 9, 10].
Indirect measures of insulin resistance and insulin sensitivity were calculated by using homeostasis model assessment (HOMA) and quantitative insulin sensitivity check index (QUICKI), respectively.
In order to assess insulin sensitivity, the following indices were calculated: homeostasis model assessment (HOMA) and quantitative insulin sensitivity check index (QUICKI).
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