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Together, our observations show that Munc18-1 and -2 promote docking by binding to closed-syntaxin-1 while a distinct interaction mode seems required to regulate the consecutive priming step.
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Among the handful of proteins implicated in basal-body/centriole duplication, centrins seem required in all eukaryotes tested, but their mode of action is not clear.
Retaliation seemed required.
Fortunately, Normal mode seems, well, more normal.
T.T.: Well, nesting mode seems to agree with your skin.
The eyebox2's Normal mode seems to be tuned toward reducing false positives, and its Close Range mode seems to be tuned toward reducing false negatives.
Initially, the mode seemed like an afterthought.
However, this mode of NMD seems to require some of the EJC proteins, RNPS1 was also dispensable (Matsuda et al, 2007).
Further research, it seems, is required.
Neuropathic pain seems to require genetic vulnerability.
However, proof of English ability seemed not required.
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