Sentence examples for mobility retardation from inspiring English sources

Electrophoretic mobility retardation assays [5] were performed using total worm extracts made by French press as previously described [19] except that 2 µg of wild-type or hcf-1 pk924 deletion mutant extract was used.

Cells were then stimulated with epidermal growth factor (EGF, 20 ng/mL) for 17 min, and BMK1 activation was detected by mobility retardation.

Whereas oligonucleotides containing the break fusion site of TMPRSS or ERG showed mobility retardation when they were incubated with the fusion polyamide, nucleotides incubated with negative control polyamide did not show a clear mobility shift.

Potential mechanistic factors used to explain the relationship between falls and psychiatric disorders include perturbations in gait, functional mobility, cognitive impairment, psychomotor retardation, changes in blood pressure and effects of psychotropic medication [ 4].

As shown in Fig. 3A, incubation of the P1882 DNA fragment with increasing amounts of CovR leads to retardation in the mobility of the DNA fragment.

MDC1 becomes hyper phosphorylated in response to IR, as demonstrated by a retardation of its mobility in SDS-PAGE that is inhibited by phosphatase treatment of the protein extracts [19], [21] [23].

Other available techniques for biomolecular interactions such as electrophoretic mobility (gel shift or gel retardation assay) and filter-binding assays provide valuable information on binding affinity.

Since reduced molecular mobility is crucial for the retardation of drug nucleation and crystallization, the selection of suitable carriers is determined by the confinement properties of amorphous drugs.

Indeed, retardation of the mobility of the RAD-51-ssDNA filaments formed in the presence of RFS-1/RIP-1 occurred when co-incubated with anti-FLAG antibodies, which bind the FLAG epitope at the RIP-1 C terminus.

Intriguingly, we found that the electrophoretic interferences on plasmid DNA are peculiar to the malten molecular structure and that CDDP, as expected (Bellon et al, 1991), causes just a slight retardation in the mobility of the supercoiled plasmid form.

Treatment of lysates from mitotically arrested cells with λ phosphatase resulted in the complete loss of the slower migrating band and a concomitant increase in the faster migrating form, indicating that the observed retardation in electrophoretic mobility was indeed dependent on phosphorylation (Supplementary Figure 4C).