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Many mobile elements exploit the existence of this recombination machinery.
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Several Integrative Mobile Elements exploiting Xer (IMEXs) were subsequently found in bacterial genomes (Das et al, 2011a), including phages (Huber and Waldor, 2002; Campos et al, 2003), cryptic elements related to phages or plasmids (Hassan et al, 2010) and genomic islands (Dillard and Seifert, 2001).
Molecular markers such as mobile elements are being developed and exploited in studies of population polymorphisms, and RNA secondary structures are used to detect signatures of selection.
Considering that there were a sufficient number of available genomes corresponding to organisms with a described presence of REP, and exploiting the advantage of the traceability of transposition events in genomes, we decided to analyze the relationship between REP sequences and mobile elements.
Mobile elements will multiply themselves, harming the individual carrying them.
Most of these genes were present on mobile elements.
These mobile elements may bias genetic relationships between isolates.
Mobile elements make up ∼45% of the human genome [1].
Accessory genetic elements mainly consist of highly variable mobile elements of antibiotic genes or mobile-antibiotic gene mosaic structures.
Mobile elements?
Considering that there are a sufficient number of available genomes with described REPs, and exploiting the advantage of the traceability of transposition events in genomes, we decided to exhaustively analyze the relationship between REP sequences and mobile elements.
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