Sentence examples for mixed inflammatory infiltration from inspiring English sources

Histological examination revealed nodular calcification in the fibrous connective tissue with dense fibrin and mixed inflammatory infiltration, predominantly with plasma cells and lymphocytes.

In a previous murine genetic model of GCP, TGF beta 1+/− mice developed glandular hyperplastic lesions that shared morphologic features with human GCP, with mixed inflammatory infiltration of the surrounding mucosa and chronic vasculitis in the tissues adjacent to these lesions [41].

Collectively, these findings suggest that young mice activate accessory molecular networks that trigger an increase in IFNγ and maintain a neutrophil-rich mixed inflammatory infiltration in the hepatobiliary system in the absence of IL-12.

Although occasional cavitary lesions were described by Driver et al. (2012), we present here further clear-cut histopathological evidence of a chronic cavity with a fibrotic capsule and mixed inflammatory infiltration with numerous neutrophils, especially in the liquefied caseum lining the cavity wall, where many neutrophils harbored acid-fast bacilli (Fig. 5).

Portal tracts revealed edema and infiltration by mixed inflammatory infiltrate.

There was moderate mixed inflammatory cell infiltration in the interstitium.

Histological examination of lung tissue include multiple, well-demarcated nodular regions of pulmonary interstitial fibrosis with mixed inflammatory cell infiltration.

NASH is histologically similar to alcoholic steatohepatitis, with the presence of macrovesicular steatosis, mixed inflammatory cell infiltration of the lobules, ballooning degeneration and necrosis of hepatocytes, Mallory body formation, and perisinusoidal fibrosis [ 7].

Histo-pathologic changes of the pectoral muscles were observed in almost 3% (n = 14) of bats and were predominantly characterized by focal or multifocal mixed inflammatory cell infiltration and single muscle fiber degeneration.

In addition, RELB deficient mice exhibited a multifocal, mixed inflammatory cell infiltration in several organs [ 53] and fibroblasts from RELB deficient mice, also indicated an important role of RELB as transcription suppressor limiting the expression of proinflammatory mediators [ 54].

In this regard, in an experimental animal model study in mice, vacuolar form of the parasite was reported to cause invasion to the lamina propria, submucosal and muscular layers of the large intestine leading mixed inflammatory cell infiltration and active colitis in infected mice [ 16].