Sentence examples for mitochondrial performance and from inspiring English sources

Thus, the carnitine shuttle system is considered to be a prominent factor in maintaining mitochondrial performance and glucose homeostasis.

Defective mitochondrial protein import likely results in suboptimal mitochondrial performance and the inability to produce sufficient energy, resulting in these growth defects (Sugiyama et al., 2007).

First, study the mitochondrial performance in large numbers of patients and controls to compensate for the genetic heterogeneity.

Based on the outcomes of our study, it is clear that defects in mitochondrial performance occur concomitantly with the decline in breeding performance, and we attribute these changes to insufficient production of CoQ by oocytes.

This modification in mitochondrial performance occurred concomitantly with an increase in whole body lipids and plasma non esterified fatty acids (NEFA), suggesting a link between skeletal muscle mitochondrial efficiency and metabolic impairments [ 10].

The investigation of the discussed selective processes and the role of the Bb, which are strictly related to mitochondrial performances, namely replication and the production and maintenance of Δψm, would also be of great importance.

In conclusion, by enhancing the mitochondrial performance, carnitine also improves the overall homeostatic response to the hypertensive insult and limits apoptotic phenomena.

Gradually researchers are changing their focus from decreased mitochondrial performance to pathogenic signaling elicited by mitochondria dysfunction (Raimundo, 2014).

Any capacity to infer the transcriptional regulation of this module alone has downstream implications for changes in mitochondrial performance.

Consequently, the protection of mitochondrial complexes by GBE may not only lead to an increase of mitochondrial performance (coupling state) but also to a decrease of ROS production.

To confirm the improvement of mitochondrial performance observed in whole cells, we investigated oxygen consumption of mitochondria that were isolated from control and APP cells after 24 h treatment with GBE.