ED transfusion may serve to reverse early tissue hypoperfusion and mitigate organ failure.
Occult hypoperfusion and mortality in patients with suspected infection.
Oxidative stress has been postulated as a major contributor to placental hypoperfusion and ischemia in pre-eclampsia (PE).
Segmental thickening parameters (systolic thickness, absolute thickening and relative thickening) appeared statistically related to the hypoperfusion and delayed enhancement scores.
The etiology of CABG-associated neurologic injury is likely multifactorial, with the phenomena of cerebral hypoperfusion and embolism being the major contributors.
Cerebral hypoperfusion and hypoxia are the main factors determining neurological outcome.
Uncorrected hypovolemia, leading to inappropriate infusions of vasopressor agents may increase organ hypoperfusion and ischemia [4].
pHi is significant parameter of tissue hypoperfusion and sympathetic nerve activity during hemorrhagic shock.
Elevated blood lactate concentrations are conventionally viewed as a surrogate of hypoperfusion and tissue hypoxia.
The sections were then evaluated for signs of hypoperfusion and ischemic hepatitis or "shock liver".
