Exact(6)
Clay minerals may also have concentrated organic compounds thereby promoting polymerization reactions.
Oil wetting of tight formations is usually controlled by adhesion to illite, kerogen, or both; adhesion to carbonate minerals may also play a role when clays are minor.
The presence of pathological minerals may also serve as a compositional marker of early-stage bone infection.
Since the stiffness of a substrate greatly affects the dynamics of actin assembly in individual cells, the enhanced stiffness of the scaffold due to incorporated minerals may also be a contributing factor to the enhanced osteogenesis.
Additional factors such as carotenoids, alpha lipoic acids, amino acids/peptides, and minerals may also offer preventive or therapeutic effects to combat hyperglycemia and several animal and culture studies have demonstrated their efficacy in improving insulin sensitivity [ 80– 80].
Alternatively, non-soil mixes made of fine bark, peat, and various minerals may also work.
Similar(54)
The release of arsenic from As-bearing minerals may be also mediated by microbial reductive dissolution.
Extraction of vital elements from the crystal lattice of minerals may be also supported by organic agents produced by the cells.
These transient increases appear to correlate with increases of soluble oxidizing products, e.g., Fe2 + and Fe3 + in solution; precipitation of secondary minerals and the formation of a passivating layer to oxidation coating the mineral surface may also contribute to these increases.
Mineral defects may also contribute to the formation of hydroxyl radicals in solution.
These results might suggest that other mineral surfaces may also have surface cations with only five neighbors.
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