Sentence examples for min before ischemia from inspiring English sources

To demonstrate the toxicity of mtDNA, mtDNA fragments were injected into rats 10 min before ischemia for 30 min and reperfusion for 24 h.

In the context of brain I/R, Kawano et al. showed in adult Mongolian gerbils that were subjected to 5-min forebrain ischemia that intraventricular injection of orthovanadate 30 min before ischemia blocked delayed neuronal death [23].

In 10 min samples, intrahippocampal perfusion of 100 nM DOPA CHE 10 min before ischemia for 70 min did not inhibit glutamate release by 10 min ischemia, while it abolished glutamate release and protected delayed neuron death by 5 min ischemia.

In the present study, HEt (1 mg/ml in 200 µl of PBS) was administered intravenously 30 min before ischemia.

Hearts were allowed to stabilize at constant pressure (60 mm Hg) 20 min prior to I/R and where indicated, TAT-Ndi1 (500 nM) was added to the perfusion buffer for 15 min before ischemia or at the onset of reperfusion.

To investigate the role of Rac1 GTPase in response to ischemic injury, 25 µg of the Rac inhibitor, NSC23766 (Tocris Bioscience, Ellisville, Missouri, USA), was dissolved in 5 µL saline and administered bilaterally into the lateral cerebral ventricles of rats 15 min before ischemia for a total dose of 50 µg.

In WT, the heart rate increased from 372 ± 22 min-1 before ischemia to 459 ± 26 min-1 (p < 0.05) after 60 min of ischemia (figure 2).

Isolated hearts were randomly exposed to different concentrations of H2O2 (1, 3, 10, 20, 30, 100, 300, and 1000  μM) for 5 min, followed by a 5-min washout before ischemia in H2O2 PC groups or for the first 5 min of reperfusion in H2O2 PoC groups.

PI3K inhibitor WM (300 nM; Millipore, Temecula, CA, USA), PKC ɛ inhibitor ɛV1 2 (10  μM; Anaspec, Fremont, CA, USA), ER stress inhibitor 4-PBA (1 mM; Sigma-Aldrich, St . Louis MO, USA), and ER stress inducer TM (2.5  μg/ml; Sigma-Aldrich) were perfused for 5 min with a 5-min washout before ischemia; hearts undergoing time-matched normal perfusion were used as controls, indicated as balance.

Baicalin was intraperitoneally injected 30 min before renal ischemia.

Intralipid group (n = 7), Group I: 30% intralipid was intravenously infused at a dose of 2 ml/kg for 30 min before renal ischemia.