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All the treatments were made 30 min before administering aspirin once daily for five days.
The animals were pretreated with GPE (30, 100 and 300 mg/kg, i.p ., vehicle (0.9% NaCl, i.p ., or indomethacin (10 mg/kg, i.p ., 20 min before administering the acetic acid [ 13, 14].
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Mice were randomized before the study and were warmed gently with a heat lamp 5 min. before administering 89Zr-DFO-trastuzumab (0.55 0.74 MBq, [15 20 µCi], 5 7 µg of mAb, in 200 µL 0.9% sterile saline) via intravenous (i.v).v
Pharmacological agents were administered 5 min before, throughout, and 10 min after desflurane administration (fig. 1).
On completion of all tasks, the symptom questionnaire and odor and environmental ratings (70 min) were administered before exposure began.
Ethanol naive mice were divided into two groups that either received saline or tandospirone (3 mg/kg, i.p ., 30 min before ethanol was administered.
Blood samples were obtained for later determination of serum glucose levels at −10 and −5 min before glucose was administered and at 30, 60, 90, and 120 min after the administration of the glucose load.
All patients received the treatment or placebo treatment for 30 min before spinal anesthesia was administered.
The trough level was taken within 30 min before a dose was administered, whereas the peak level was taken 1 h after the 5 min i.v. infusion was started.
Hoechst 33342 and DiOC7(3) were either administered simultaneously or Hoechst 33342 was administered 20 min before DiOC7(3).
Glulisine was administered immediately before meals, regular insulin was administered ∼30 min before meals.
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