Thus it is theoretically possible that humans reporting mild sequelae following monkey bites and scratches might be describing a mild variant of B virus syndrome.
At 46 and 53 dpi, this monkey showed mild portal infiltration, and at necropsy, the liver histopathology was scored as 1 using the severity assessment scoring system for inflammatory lesions (Table 2).
A potential live-attenuated EV71 vaccine candidate (S1 3′) developed by the Shimizu's group in Japan had been shown to elicit a broad spectrum of neutralizing antibody responses against different genotypes of EV71 in cynomolgus monkeys, but mild neurological symptoms were also observed.
All monkeys in TA group had a mild interstitial pneumonia, and 4 monkeys in TB group had a mild to moderate interstitial pneumonia.
In the TC group, one monkey had a mild to moderate interstitial pneumonia, and other 3 monkeys had a moderate to severe interstitial pneumonia.
First, adoptive transfer of autologous LCV-infected B cells induced autoreactive T-cell activation and (mild) meningeal inflammation in rhesus monkeys 25 and marmosets.
These monkeys developed a mild dyskinetic syndrome by the end of the first month of treatment, then displaying overt dyskinetic symptoms 1 month later and remained stable until the CTB retrograde tracer injection.
Fornix transection therefore produced a very mild impairment in DNMS in all monkeys, but this effect did not differ whether the monkeys had cholinergic depletion of inferotemporal cortex or not.
Third, fornix transection produced a mild impairment in DNMS in both control monkeys and in monkeys who had received cholinergic lesions of inferotemporal cortex, but the magnitude of the impairment was not increased in monkeys with cholinergic depletion of inferior temporal cortex.
But unlike monkeys, mice only develop mild disease from Ebola strains, so the experiment relied on a mouse genetically engineered to lack immune defenses against viruses.
