Sentence examples for mild migration from inspiring English sources

Despite the relatively mild migration defect caused by bbg knockdown, partially migrated bbg RNAi border cells exhibited reduced nuclear Stat92E and presumably reduced JAK/STAT activation.

This suggests that loss of 95% of FLNa expression is sufficient to produce a mild migration phenotype but that the additional loss of FLNb is required to produce a more robust effect, presumably because FLNb compensates for the lack of FLNa [25].

We observed that 62% (n=88) of double heterozygous scrib +/rw468; nhsl1b +/fh131 embryos exhibited an almost complete loss of FBM migration, compared with much milder migration defects in only 8% (n=85) and 18% (n=69) of single nhsl1b +/fh131 or single scrib +/rw468 heterozygotes, respectively (Fig. 3A-C).

The authors conclude that angioblast migrate normally in apelin mutants, however, in Figure 2 it appears that ca. 30% of apelin mutants showed mild angioblast migration phenotype.

In contrast, deletion of the cytoplasmic domain of β-DG did not result in disruptions of the glia limitans while displaying a mild cerebellar migration defect [ 7].

We previously showed that co-expression of Ben and dUev1a resulted in JNK activation; consistently, Ben and dUev1a expression along the A/P boundary induced a mild cell migration phenotype and upregulation of MMP1, which was significantly enhanced by removing one copy of endogenous puc, indicating that Ben/dUev1a expression could induce JNK-dependent cell migration.

At P10, the majority of miR-Vps35 transfected neurons had migrated to pyramidal cell layer of hippocampal CA1 region, however, a mild but significant migration defect was observed in miR-Vps35-1 neurons: ∼13% of neurons were mislocated out of pyramidal cell layer as compared to ∼5% in control (supplementary material Fig. S2).

The strategy is based on the combination of a regioselective hydrolysis catalyzed by immobilized lipases with a subsequent mild chemical acyl migration.

The histological examination of the arthritic joints treated with indomethacin exhibited mild inflammatory cell migration.

However, knockdown of each of these genes only led to a mild decrease in migration in scratch wounds.

Knockdown of positive hit PDZ genes resulted in mild to moderate migration defects, with most border cells able to detach from the epithelium and migrate partway to the oocyte.