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Migratory rate of both cells was significantly increased at even low normal levels of UA.
Exposure of human mammary cancer cells or mouse mammary epithelial cells in vitro to a wide concentration range of UA dose dependently increased migratory rate of both cells[136].
Intriguingly, our NSCs displayed a significantly higher migratory rate towards tumor cells under hypoxic condition.
MCF-7 cells overexpressing BST-2 had higher migratory rate compared to vector control cells.
However, LMP1 enhances the migratory rate of Jurkat cells subsequent to invasion of the extracellular matrix, and Fascin accounts primarily for this phenotype.
In this case, MIH55 cells had an increased migratory rate compared with the PAP60 cells and resembled the migration capability of HT29 cells (Table 2).
Similar(53)
Lastly, the number of N-glycans associated with the Kv3.1 protein influenced neuroblastoma cell migratory rates.
Using an in vitro scratch wound assay we demonstrate that the genetic variants in the KRT1 haplotype block are significantly associated with differences in the migratory rates of wound-activated HEK cells.
The wound healing versus time curves demonstrate that B35 cells expressing glycosylated Kv3.1 have faster migratory rates relative to those expressing unglycosylated Kv3.1, and non-transfected B35 cells, as well as the partially glycosylated Kv3.1 proteins.
In this study, it was shown that migratory rates of the neuroblastoma cells were altered by the absence of 1 or 2 N-glycans of the Kv3.1 protein relative to that with both N-glycans.
We demonstrated that B35 cells expressing the unglycosylated Kv3.1 protein had outward ionic currents with slower activation and deactivation rates, and reduced cell migratory rates compared to those of the glycosylated Kv3.1 proteins, indicating that the N-glycans of the Kv3.1 glycoprotein influence conducting and non-conducting roles of the Kv3.1 channel.
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