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By using an affinity column constructed by immobilizing E-selectin/Fc recombinant, the E-selectin binding protein that mediated breast cancer cell transendothelial migration was identified as human CD44 (Fig. 4).
But in CHAC1 knockdown BT-20 cells only a tendency of a reduced migration in vitro was observed, whereas in CHAC1 overexpression BT-20 cells an increased migration was identified.
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Unique clinical characteristics of IUD migration were identified in patients with uterine perforation.
Important artefacts associated with surface oxidation and grain boundary migration were identified by removing surface material with a focused ion beam (FIB) to examine the microstructure beneath.
Specific mechanisms for unanticipated CO2 and brine migration were identified with both established and novel mitigation approaches for remedying them proposed.
Bands or spots were chosen by Western blotting, then proteins with equivalent migration were identified from another gel.
The PI3K/PAK1/ERK signaling pathway for LPA-stimulated breast cancer cell migration is identified and LPA is found to stimulate ROS generation in breast cancer cells.
It took 2 years until endogenous EBI2 agonists that control B-cell migration were identified.
Deaths and migration were identified in the routine reenumeration within the KHDSS during the follow‐up period.
All relevant codes are provided in Additional file 1. Deaths and migration were identified from the Danish Civil Registration System through 31 December 2011.
Subgroups of shared genes, also predominantly related to cell adhesion and migration, were identified in pairwise comparisons of ECC, limb buds and PNST cells.
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