Sentence examples for migration of peripheral from inspiring English sources

While mechanisms for efficient humoral responses to lymph-borne antigens have been characterized, the current paradigm for T cell responses to infections and particulate vaccines involves delayed migration of peripheral antigen-bearing dendritic cells (DCs) to lymph nodes (LNs), where they elicit effector T cell responses.

In contrast, in animals infected with L. donovani, the migration of peripheral blood monocytes to hepatic granulomas [33], which is under the control of GM-CSF, generates leishmanicidal activity throughout macrophage activation [34].

This suggests that the long held paradigm that coordinated maturation and migration of peripheral DC to the lymph node is required for T cell activation may be more complicated than first described.

Natalizumab blocks the interaction of these molecules with their receptors, vascular-cell adhesion molecule 1 (VCAM-1) and mucosal addressin-cell adhesion molecule 1, present on the vascular endothelium resulting in decreased migration of peripheral inflammatory cells into the target tissues [2], [3].

First, we analyzed the effects the chemokines on migration of peripheral blood B cells.

Although CX3CL1 induced robust migration of peripheral NK cells in vitro, this may not reflect the action of this drug in vivo.

We wondered whether this pattern resulted from limitations of lymphatic entry [45] or migration capacities of peripheral DCs [46].

Furthermore, LL-37 induces migration of human peripheral blood monocytes, neutrophils, CD4+ T cells, and mast cells [12], [13], [20], [21] and influences the expression of over 40 genes in murine RAW 264.7 macrophage cells, in which some of these gene products are involved in bone formation, including the bone morphogenetic protein (BMP) 1, BMP-2, and BMP-8a [22].

Migration of cultured peripheral blood mononuclear cells (PBMCs) from Medalists and age-matched controls were compared.

Migration of human peripheral blood monocytes induced by monocyte chemotactic protein-1 (MCP-1) or supernatants of different tumour cell lines under the influence of VALE or OA was measured in a chemotaxis transmigration assay.

To determine the extent to which increased trafficking played a role, we examined the effect of ethanol on the migration of different peripheral blood mononuclear cell (PBMCs) subsets across a brain endothelial cell monolayer.