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Shin, S. H. et al. Implication of leucyl-tRNA synthetase 1 (LARS1) overexpression in growth and migration of lung cancer cells detected by siRNA targeted knock-down analysis.
Wang J, Weng Y, Zhang M, Li Y, Fan M, Guo Y, et al. BMP9 inhibits the growth and migration of lung adenocarcinoma A549 cells in a bone marrow stromal cell-derived microenvironment through the MAPK/ERK and NF-κB pathways.
Remarkably, miR-423-3p miR-423-3p miR-423-3p lung cancer cells in vitro.
Our experiments show that TDP-43 promotes migration of lung cancer cells by binding to and regulating miR-423-3p miR-423-3p miR-423-3p
Our results suggest that TDP-43 may promote migration of lung cancer cells by regulating miR-423-3p miR-423-3p miR-423-3p
In the present study, we reported that miR-19a and miR-19b (miR-19a/b) promote proliferation and migration of lung cancer cells by targeting MTUS1.
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Wang et al., showed that elevated serum RbAp46 levels are highly correlated with non-small cell lung cancer (NSCLC) distant metastasis and knockdown of RbAp46 inhibits the migration ability of lung cancer cells [ 20].
Specifically, in breast and lung cancer cell lines (A549, MDA-MB-468, MCF-7), treatment with nicotine increased the proliferation, invasion, and migration of both lung and breast cancer cells in a dose-dependent fashion.
Slit treatment led to a significant reduction in cell migration of H1299 lung cancer cells.
Silencing of the uch l1 gene is reported to reduce migration of H157 lung cancer cells[11].
We have recently shown that S1P signals through S1P1 and Gi to activate PKC-ε and subsequently, a PLD2-PKC-ζ-Rac1 cascade to induce migration of human lung ECs [12].
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