Sentence examples for migration of epidermal from inspiring English sources

Re-epithelialization is an essential step of wound healing involving the migration of epidermal keratinocytes over the wound site.

These ROS result in membrane lipid peroxidation, protein oxidation and damage to nucleic acids, any of which may impair cellular processes involved in wound healing including proliferation and migration of epidermal cells, and angiogenesis [18].

However, our model analysis indicated that cell proliferation is equally as important as cell migration (as other reports have suggested [11], [58], [64], since an extra source of keratinocytes is crucial in wound healing for the continuous migration of epidermal cells as a cohesive sheet.

Most importantly, HGF improved the proliferation and migration of epidermal cells.

The proliferation and migration of epidermal cells are crucial for the rapid closure of the epidermis [ 1, 2].

Bandyopadhyay and colleagues discovered a key factor in human serum, TGFβ3 that seems to play the "traffic control" role in migration of epidermal versus dermal cells.

The influence of the block on migration rule or hyperproliferation rule on epidermal wound healing was also evaluated by measuring the migration distance of epidermal cells and the percentage of the wound area that was covered by cells as shown in Fig. 6A B.

Silencing of ILK gene expression by RNA interference reduced expression of β1-integrin, ILK, and c-met in epidermal cells, concomitantly decreasing the proliferation and migration ability of epidermal cells.

In this study, silencing ILK gene expression by RNA interference decreased the expressions of β1-integrin and c-met, concomitantly reducing the proliferation and migration ability of epidermal cells.

Healing commences after the formation of a fibrin clot that is invaded by granulation tissue and by the migration of an epidermal tongue along the interface between the granulation tissue and the clot (schematic in Figure 1A).

In a recent study, migration of human epidermal keratinocytes, in in vitro scratch wounds, was inhibited by LPS and the inhibition could be blocked partially by anti-TLR4 (75%) and anti-TLR2 (40%) [ 26].