Sentence examples for migration of astrocytes from inspiring English sources

She studied the migration of astrocytes, cells in the central nervous system that are "very underestimated and understudied and probably play an important role in many pathologies".

What is the mechanism by which sustained Ro3303544 stimulates the migration of astrocytes?

Such a conserved function of Wnt has also been identified during directed migration of astrocytes (Schlessinger et al., 2007).

Since our proposed mechanism for the enhanced migration of astrocytes upon Ro3303544 administration implies the presence of high ECM concentrations, the changes in laminin expression, as an example ECM protein, were examined following SCI.

Not surprisingly, the PRG-A list contained genes primarily involved in cellular proliferation and growth, cellular migration and inflammatory disease, accurately reflecting the proliferation, activation, and migration of astrocytes and glial cells to damaged areas of the brain.

Glioblastoma cells infiltrate brain tissue and migrate preferentially along white matter fibre tracts, an environment that is highly inhibitory to the migration of astrocytes and the growth of neurites because of the presence of specific inhibitory proteins.

Quantitative analysis revealed no significant difference in the number of BrdU-labelled reactive astrocytes surrounding the lesion, implying that the increased compaction of infiltrated inflammatory cells by Ro3303544 results from the migration of reactive astrocytes rather than from astrocyte proliferation (Fig 4D).

This treatment resulted in accelerated migration of reactive astrocytes to sequester inflammatory cells that spared myelinated fibres and significantly promoted functional recovery.

Administration of Ro3303544 for the first 5 days after contusive SCI resulted in faster migration of reactive astrocytes, preservation of more myelinated fibres, and significantly improved recovery of movement.

Given the complexity of the lesion environment, as well as the number of molecules that potentially modulate the migration of reactive astrocytes, it seems plausible that the actual mechanism for the in vivo enhancement of migration by Ro3303544 is more complex than our proposed model.

By using short- and long- chambers where CSF-intake holes are partially or totally exposed to the CSF, the system models migration of activated astrocytes and microglia out of the brain onto the shunt extending into the ventricle when the catheter is sub-optimally placed, or when placed in its intended location.