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The logic of the family migration model suggests that source country gender roles within the family might be a factor in influencing the shape of women's assimilation profiles, with the family migration model perhaps holding for families coming from countries with a more traditional division of labor by gender.
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Our model suggests that discouraging migration might provide an alternative means of cancer suppression.
This model suggests that the brain-migration association is in part caused by unanalyzed effects [sensu 36] associated with the correlation between BMR, incubation period and body mass, once latitude is taken into account.
In contrast, the second model suggests a multidirectional route: one migration through central Asia and one through southeast Asia [1], [5], [6].
First, our model suggests that we should find greater expression of migration related proteins in neoplasms with regions of hypoxia compared to neoplasms with uniform oxygenation.
But Dr. Hall's model suggests otherwise.
The model suggests two independent predictions.
The model suggests that TGF-β1 plays a crucial role in normal re-epithelialisation by dictating the balance between cell proliferation and migration.
The dominant mammalian model suggests ∼2 glomeruli/OR.
An alternative model suggests the largest ancestral contribution to ISEA populations results from a third and more recent event, which is associated with one of the largest agriculturally-driven migrations in human history: the Neolithic expansion from Taiwan.
Thus, the model suggests that the differential ability of CCL19 and CCL21 for desensitizing CCR7 combined with the hypothesized physiological configuration of superimposed CCL19 and CCL21 fields (possibly in the periphery of TCZ) enable the repulsive migration of T cells.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com