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Reduced estrogen biosynthesis or suppression of tumor necrosis factor-α and leukocyte migration may contribute to the tocolytic actions of dexamethasone and interleukin-10, respectively.
Although the full characterization of the mechanisms of Rac1 activation and its consequences in oral keratinocytes may be beyond the scope of the present study, these observations prompted us to explore whether the ability to control gene expression programs, in addition to cell migration, may contribute to the healing defects caused by Rac1 ablation.
Such findings suggest that disruptions in neuronal migration may contribute to the etiology of language-based disorders.
DC migration may contribute to the common pathological finding of sinus histiocytosis, producing enlargement of regional, draining, non-tumour involved lymph nodes in primary breast (and other) tumours.
The findings suggest that BK-induced MMP-9 expression and cell migration may contribute to increase BBB permeability and recruit immune cells, leading to brain inflammation and edema.
Moreover, our in vitro scratch assay suggests that both fructose and MG-induced SMC migration may contribute to the neointimal formation and the inhibitory effect of metformin on SMC migration could contribute beneficial effects by reducing stenosis.
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Since migration may take a non-trivial amount of time, migration itself may contribute to, or even cause, an SLA violation.
In sum, the existing evidence suggests that migration experience may contribute to shape migrants' political attitudes, in a way that has been found to depend on several factors.
Thus, oncogenic PI3K/mTOR activation promotes cytoplasmic p27 accumulation, increased cell migration, and may contribute to neoplastic progression.
It also attempts to identify factors, such as operation size, hive migration, etc., that may contribute to losses in general, and CCD in particular.
Since enhanced migration and invasion may contribute to an increased metastatic potential of CT45-expressing cells, this might in part explain the more severe disease progression and poorer prognosis in patients with CT45-positive tumors.
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