Sentence examples for migration in well from inspiring English sources

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We previously reported that normal fibroblasts (NFs) and, notably, breast cancer-associated fibroblasts (CAFs) induced epithelial-to-mesenchymal transition and increases in cell membrane fluidity and migration in well- (MCF-7) and poorly-differentiated (MDA-MB-231) breast cancer cells.

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LC3 deficiency significantly inhibited MDA-MB-231 cells migration in transit well chamber as well as invasion in matrigel coated transit well chamber assay (Supplementary Figures 7 and 8).

After ageing at 50 °C and 70% RH, there is evidence of silver migration in the well processed photographs on all paper types.

Migration in control wells, in which FBS was replaced with 0.1% BSA was minimal (%migration <2.4±1.5%).

The cell line with negative expression of fascin displayed the weakest migration in trans-well test, and the cell lines with high and moderate expression of laminin-5γ2 displayed stronger migration phenotypes than those with negative expression of laminin-5γ2.

Delta9-Tetrahydrocannabinol inhibits epithelial growth factor-induced lung cancer cell migration in vitro as well as its growth and metastasis in vivo.

In turn, through a kinome-wide phosphoproteomic and MS study, we demonstrate that this scaffolding protein regulates a shift in protein kinase A (PKA -mediated PKA -mediatedon events under hyphosphorylationalteventsns in tundercell invasion and migration in vitro, as well as metastasis in an in vivo orthypoxia model of melanoma.

Dr. Liechti's research focuses on quantifying the general patterns in bird migration in relation as well as investigating the individual patterns and flight behaviour of self-powerd Trans-Sahara migrants.

While studies on the evolution of migration in birds are well represented in the literature, migration in bats has received relatively little attention.

Marchetti et al. have now shown that DPP-IV inhibits HMGB1-induced endothelial cell migration in vitro as well as HMGB1-induced neovascularisation in vivo [ 5].

The pharmacological GPR55 inhibitor CID16020046 inhibited LPI-stimulated ECFC proliferationetworkork formation and migration in vitro as well as ovarian carcinoma cell- and LPI-induced angiogenesis in vivo.

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