Sentence examples for migration in intestinal from inspiring English sources
The high affinity LTD4 receptor CysLT1R exhibits tumor-promoting properties by triggering cell proliferation, survival, and migration in intestinal epithelial cells.
Nevertheless, loss of FHL2 activity is associated with increased epithelial cell migration in intestinal epithelium, which might allow to eliminate more efficiently deleterious cells and reduce the risk of tumorigenesis.
By employing a genetic approach in vivo, we demonstrate that loss of FHL2 drastically suppresses tumorigenesis in the murine intestine, which may be partly due to the positive effects of FHL2 deficiency on cell migration in intestinal epithelium.
The kinetics of cell proliferation, differentiation, and migration in intestinal epithelium are described.
A fish hydrolysate preparation has been shown to induce proliferation and migration in intestinal epithelial cells, which may contribute to anti-inflammatory and healing properties [ 13].
To determine the role of PGP in neutrophilic migration in intestinal inflammation, we investigated these components and the effects of PGP neutralisation in mice with DSS-induced colitis.
Cell migration and adhesion are essential in intestinal epithelial wound healing and recovery from injury.
We observed similar results regarding the contribution of CD47 and COX-2 in intestinal cell migration in both a 2-dimensional assay on a collagen I coated surface and in a 3-dimensional assay that required migration through a collagen I containing gel.
Such disease states as ulcerative colitis, Crohn's disease and infectious enterocolitis, involve neutrophil migration across intestinal epithelia, neutrophil accumulation in the inflamed intestine [24], [25], overproduction of inflammatory cytokines, and release of massive amounts of ROS from neutrophils [26], [27].
The loss of FHL2 activity in intestinal epithelium clearly accelerates cell migration, which constitutes a fundamental mechanism in the control of tissue homeostasis.
These studies were supported by in vitro models of transepithelial migration and intestinal epithelial barrier function.
