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Since enhanced cell migration in cell chamber systems is reported to correlate with increased invasive properties in vivo, the presented inhibitory effects of PACAP 38 on neutrophil chemotaxis, supports the idea of an anti-inflammatory role for PACAP.
On the basis of our results, we found that the most important factors influencing the cell behaviors on the three sets of solid substrates are the degree of dimension (2-D or 3-D migration) in cell behaviors and the cell traction force.
There is a modest increase in cell migration in cell lines overexpressing miR-30a compared to their controls.
Nonetheless, VT reduced trans-endothelial permeability and tumor cell migration in cell culture.
We previously demonstrated that TRAM-34 is effective in reducing CXCL12-induced GBM migration in cell lines and primary cells obtained from patients.
Epidermal keratinocytes extracted from Map3k1 ΔKD mice have defective Tgf- β, Activin and Activin B-inducedced migration in cell culture plate-based wound healing assays.
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We sought to determine how TGF-β affects tumor cell migration in cells grown in different collagen concentrations and for different incubation times.
In effect, CUL2 migration in cells expressing UBXN7 ΔUIM was similar to untransfected cells.
Growth receptors, when interacting with corresponding ligands, initiate the process of cell cycle progression and migration in cells.
There were no significant differences in speed of migration in cells treated with control or Vav2 siRNA (Fig. 4C).
In this study, we observed that TSG101 depletion bidirectionally modulated cell invasion but not cell migration in different cell types.
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