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Scanning of the fracture surface at the end of the migration experiment showed limited mobility of α-emitting radionuclides and of the rare-earth elements, consistent with static sorption data obtained on representative fracture surface material.
Migration experiment showed that more MDSCs went through a transwell membrane toward the media with G-CSF, which indicated that G-CSF could modulate the movement of MDSCs (Fig. 4A).
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In addition, our cell migration experiments showed that a higher CRN5 expression level causes an increased tumour cell migration velocity.
Transwell migration experiments showed that normal human lung fibroblast (NHLF) transmigration through collagen I-coated inserts is mediated by DDR2 and the DDR2-associated signaling kinases JAK2 and ERK1/2, but not DDR1.
The transwell experiment showed that migration of TSGH8301 bladder cancer cells was considerably suppressed by c-Met siRNA knock-down (p < 0.001).
In contrast to that observed in the in vitro migration experiments, HUCPVCs showed a similar migration capability towards nontumoral tissues like liver or lung and also showed a lower recruitment to the spleen.
The BrdU pulse-chase experiment showed that blocking FGFR1 reduced cortical migration of new born cells and their differentiation to neurons.
To determine the effect of overexpression of Pfn1 in cell motility, we first performed transwell migration experiments which showed that overexpression of GFP-Pfn1 suppresses chemotaxis of MDA-MB-231 cells by nearly 80%.
The transwell experiment showed that Fas stimulation of uninfected and HSV-2-infected keratinocytes induced a significant migration of Gr-1-positive cells, in comparison with migration toward control, uninfected cells (P≤0.001).
This experiment showed 0.9% mutations.
Compared to the original live migration, our experiments show that the optimized scheme can reduce up to 88% of application downtime with an acceptable overhead.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com