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MRP8/14 – a marker for transendothelial migration – describes not only the state of inflammation in diabetic nephropathy, but additionally the degree of microvascular alterations in the glomerular and retinal bed.
In summary, we demonstrate that MRP8/14 as marker for transendothelial migration describes not only the state of inflammation in diabetic nephropathy, but additionally the degree of microvascular alterations.
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Cell migration is described using not only haptokinetic, but also chemotactic and haptotactic influences.
However, migration was not described being a risk factor for NSSI in these studies, due to the fact that it had not been included as a possible risk factor in most studies.
In mice, IL-8, the prototype of human CXC chemokines that mainly affect neutrophil migration, has not been described, but the only neutrophil-attracting chemokine recognizing CXCR1 and 2 is GCP-2, generally considered as the functional equivalent of IL-8 in humans [ 46, 47].
In the first 2 hours after wounding, untreated cells showed negligible rates of migration, as described previously, and addition of 10 μM U0126 did not change that.
Interestingly, in a study on Boston youth, Borges et al. [ 15] described adolescents with a migration background not to be at higher risk for NSSI and suicidal ideation than US born youth.
Migration was not a word I would have used to describe what I was doing when I sailed with other West Indians to England in 1950.
SU5402 and PD173074 induced the same phenotypes during primordium migration (data not shown) as well as intercalary neuromast formation described in this paper.
While many researchers use the term "migration intention" to describe future possible migration behavior, we use the term "migration desire" since the main survey question we used is not about planned actions.
Describing the differences in Western and Gulf migration policies, Mr. Janardhan, the Emirates-based political analyst, explained how the term "migration" did not apply to expatriates.
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