Sentence examples for migration and prevent from inspiring English sources

We have shown that in normal wound healing FOXO1 functions to reduce oxidative stress in keratinocytes that is necessary to maintain cell migration and prevent cell death [ 19].

FOXO1 also functions in keratinocytes to reduce oxidative stress that is necessary to maintain cell migration and prevent cell death in a TGF β1 independent manner.

Thus, the reduced expression induced by milatuzumab in vivo may impair cell migration and prevent their proper participation in immune responses.

The microporous membrane with a small distance to the plug provided a geometric barrier to inflammatory cell migration and prevented leukocyte-mediated degradation of the plug for at least 30 days.

In vitro, Sprouty-4 inhibited fibroblast growth factor and vascular endothelial cell growth factor-mediated cell proliferation and migration and prevented basic fibroblast growth factor and vascular endothelial cell growth factor-induced MAPK phosphorylation in endothelial cells, indicating inhibition of tyrosine kinase-mediated signaling pathways.

By activating the VEGF Receptor-2, low-dose IR enhances endothelial cell migration and prevents endothelial cell death promoted by an anti-angiogenic drug, bevacizumab.

By activating VEGFR-2, low-dose IR enhances endothelial cell migration and prevents endothelial cell death promoted by the VEGF-neutralizing antibody bevacizumab.

Taken together these results demonstrate that low-dose IR enhances endothelial migration and prevents endothelial cell death promoted by the VEGF-neutralizing antibody bevacizumab, by inducing VEGFR-2 phosphorylation.

This paucity of information limits our understanding of the evolution of migration and prevents us from asking such questions as: are the same genetic mechanisms involved in migration conserved across different taxonomic groups?

IFN-β downregulates the proinflammatory cytokines IL-1β and tumor necrosis factor α (TNF-α) and enhances IL-10 and IL-1 receptor antagonist production by lymphocytes in vitro [ 4- 6], increases IL-1 receptor antagonist production by fibroblast-like synoviocytes (FLS) [ 7], inhibits T-cell proliferation and migration, and prevents contact-dependent T-cell activation of monocytes [ 8].

f) Immune evasion mechanisms are increased: MAN1A2, (which induces T-reg cell migration and prevents T cell priming [ 62]), was increased and this supports our previous contention that as neoplastic transformation proceeds, a T-reg-like phenotype is induced [ 3, 6, 12].