Sentence examples for migration and generation from inspiring English sources
Hemoadsorption with AC reduced serum concentrations of resistin and restored neutrophil migration and generation of ROS to normal levels.
Here, incubation of NB4PMN cells in resistin-spiked serum after Amberchrome CG161M™ treatment restores both transwell migration and generation of ROS back to control levels.
While no resistin receptor has been identified in humans, resistin is known to interfere with intracellular signaling involving the phosphatidylinositol 3-kinase (PI3K) pathway which is crucial for key neutrophil functions, including migration and generation of ROS [14].
With each succeeding migration and generation, improvements are made and progress becomes discernible.
In the present study, we investigate alterations in kinin receptor expression on angio-supportive circulating cell types, namely CD34pos CPC and monocytes, and kinin-induced cellular functions, such as migration and generation of O2−* and NO, in type 1 diabetic patients (T1D) prior to the onset of cardiovascular disease.
Calycosin improved cell viability, suppressed apoptosis and protected the cells from LPS-induced reduction in cell migration and generation of ROS, protein level of NOS at a comparable magnitude to that of Y27632 and valsartan.
We further tested AC for their effect on cell migration and ROS generation and CC for their effect on bacterial clearance.
Resin adsorption therapy with Amberchrome GC160M™ successfully removed resistin from resistin-spiked serum samples and restored both impaired neutrophil migration and ROS generation to normal levels.
Neutrophil recruitment and migration were analyzed by transendothelial migration and ROS generation.
In conclusion, this study demonstrates that the 12(S -HETE-induced intravaS -HETE-induced S -HETE-inducedgh LECs requintravasation-dependent profess of LECs triggering the disintegration of cell–cell contacts, migration, and the generation of CCID.
Little is known about the role of TGFβ3 in later involution, though it has been hypothesized to promote fibroblast migration and ECM generation based on the upregulation of wound healing and ECM genes that are targets of TGFβ signaling during this phase [ 17, 63, 65].
