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Breast cancer metastasis depends on cell migration, a complex process regulated spatially as well as temporally by the Rho family GTPases Rho, Rac and Cdc42 [1].
Migration, a complex phenomenon, has long held centre stage in discussions concerning the human resources for health crisis.
Migration, a complex multi-step process, starts with the formation of an internal cellular asymmetry of molecules and structures that allows the establishment of a front rear cell polarity that eventually translates itself in a directional movement towards the site of interest (Bornens, 2008; Li and Gundersen, 2008; Mellman and Nelson, 2008; Stramer et al., 2010).
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Migration is a complex process that involves coordinated polymerization, adhesion, actin polymerization, and movement to the membrane in the migration direction.
Migration is a complex subject.
Background: Cell migration is a complex phenomenon that requires the coordination of numerous cellular processes.
Grade migration is a complex phenomenon, which in reality will depend upon details of the detector, the incident spectrum, etc.
Cell migration involves a complex, multistep process that leads to the actin-driven movement of cells on or through the tissues of the body.
The damage reduction with varying composition is further ascribed to the increasing heterogeneity of point defect migration across a complex potential energy landscape that enhances defect recombination.
Migration is a complex subject to approach in archeology and the new materials and methods available, such as isotope analysis and DNA, make it possible, and necessary, to ask new questions.
Cell migration is a complex process, requiring coordination of many subcellular processes including membrane protrusion, adhesion, and contractility.
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