Based on the results above, we conclude that the apical-to-basal nuclear migration that occurs in G1-phase is subject to a displacement or crowding-out effect of incoming nuclei migrating in the opposite direction after exiting S-phase.
In addition, we observed that immature DCs migrating in confined micro-channels display important velocity fluctuations, alternating between fast and slow motility phases (Faure-Andre et al, 2008).
The macromolecular assemblies related to the specific ligands were then separated using a differential microdialysis system, based on liquid transitory pH gradient (pH 5 10) where the macromolecules (protein groups) can migrate in the liquid phase to their mean molecular zwitterion positions51,52,53.
In contrast, the H2S in the condensates of the Yingshan Formation (O1y) may have not dissolved and migrated in the gas phase.
Considering the short time window of our observation (max 3 hours after the induction of ischemia), we cannot exclude that NCs could migrate in a later phase from the MAP-2 positive penumbra regions towards the MAP-2 negative ischemic zone.
Alkali chlorides were observed to migrate in the gas-phase towards the colder temperatures.
Free phenolics migrated in this last organic phase, which was then evaporated to dryness and redissolved in methanol for further analyses.
However, whereas only 0.05% of RINs migrated >5 µm tangentially, which is roughly one somal length, during Phase 1, 27% of RINs migrated >5 µm in Phase 2 and 22% of RINs migrated >5 µm in the first 12 h of Phase 3, suggesting that tangential migration occurs mainly during later stages of migration.
This suggests a role for Sma in migration only during the vegetative-growth phase, when cells migrate in search of bacteria, which they locate via folic acid.
