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Moreover, the WRKY and Myb TFs and LRR-VIII and SD-2b kinases might regulate downstream genes under FA stress but not general allelochemical stress.
Our results suggest that WRKY and Myb TFs and LRR-VIII and SD-2b kinases might regulate downstream genes under FA stress but not general allelochemical stress.
Among the 1,285 DEGs from the two tissues, we identified 91 transcription factors, which might regulate downstream components of exercise-triggered signaling pathways (Additional file 1: Table S19).
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This was the first indication that TOR, actually TORC1, might regulate growth downstream of nutrient cues.
Next, we reasoned that if L148S could interact with β-arrestin-1 and 2, it might mean that GPR54 molecules could regulate downstream signaling events like gene expression in the absence of Gq/11.
This modulation might run through two ways: (1) regeneration genes can regulate downstream genes and pathways that are already known to be associated with them; (2) regeneration genes could regulate completely novel genes and pathways.
On an upstream level, Histone proteins regulate downstream gene expression, and zinc finger proteins might modify those in turn.
Secondly, changes in leptotene chromosome architecture regulate downstream meiotic processes.
The cellular adaptations to sustained hypoxia are mediated by hypoxia-inducible factors (HIFs) to regulate downstream target gene expression.
In hypoxia, stabilized HIF-1α and HIF-2α bind to ARNT to regulate downstream target gene expression1,2,5,6.
PcGs epigenetically regulate downstream target genes [ 28].
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