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Therefore, CaR might likely mediate the mechanism of insulin secretion.
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Important components of this transition are likely mediated by toll-like receptors, neuroinflammation, and tauopathy.
Taken together, the preservation of juvenile OD plasticity in rl−/− mice is not likely mediated by changes in inhibitory tone, suggesting that other functional and/or morphological changes might be responsible for enhanced and prolonged plasticity in rl−/− mice.
This mechanism is most likely mediated by crosstalk between IR regulators (Senn et al., 2003).
In one study an antinociceptive effect was found after 5 30 mg/kg, most likely mediated by the 5-HT1A receptor [28].
These effects were likely mediated by NADPH oxidase induced oxidative stress inside the cell[61, 62, 65, 66].
They suggested that biotic Mn oxidation was most likely mediated by a phylogenetically diverse microbial community (Nitzsche et al. 2015).
This result is most likely mediated by the ability of fibrinogen to enhance fibroblast migration [54, 55].
These processes are most likely mediated through mitochondria-originated ROS.
This is most likely mediated through nutritional status.
The inhibitorial effect is likely mediated through a diffusible agent.
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