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A general property of viral infections might cause increasing accumulation of host gene products as part of a stress response, especially HSPs genes.
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Besides osteoarthritis there are various conditions which might cause increased bone turnover and uptake on bone scan like older trauma, benign bone lesions like enchondroma or osteonecrosis.
Microvascular edema might cause increased barriers for oxygen diffusion with reduction of cellular oxygen delivery despite the absence of frank cerebral ischemia [17].
Asynchrony might cause increased values when measurements are made gated by ECG.
Therefore, it is possible that the decline of suPAR excretion with decreased GFR might cause increased serum levels.
Our study found that increased exposures to PM2.5 might cause increased acetylation levels of H3K27ac markers for specific genes involved in platelet activation, blood coagulation and hemostasis.
In turn, these reactive species might cause increased lipid peroxidation in neuroglial membranes, thereby accounting for the observation of increased ethane excretion.
Microvascular edema might cause increased barriers for oxygen diffusion with reduction of cellular oxygen delivery despite the absence of frank cerebral ischemia [ 17].
Absence of centromeric heterochromatin might cause increased kinetochore flexibility, making it more prone to interaction with microtubules coming from opposite poles.
We hypothesized that the different current patterns might cause increased expression of β-Gal because DNA uptake is proportional to current concentration.
Moreover, MSI-H tumors might cause increased immune reactivity as a consequence of the high amounts of aberrant frameshift peptides they generate [ 8, 18].
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