In conclusion, we demonstrated that α5 expression levels on tumour cells might affect kidney metastasis rates.
> -wrap-foot> We also evaluated the possible risk factors that might affect nodal metastasis.
Clues for the mechanism by which αB-crystallin might affect distant metastasis formation in patients were obtained by the depletion of αB-crystallin in HNSCC cell lines, which showed that both VEGF secretion and cell motility were decreased when αB-crystallin expression was reduced.
Because the 'cell death' signature contained some genes that also had roles in processes that might be expected to affect metastasis, i.e. cell migration and invasion, we assessed the prognostic ability of genes with a 'cell movement' annotation compared with the remainder of the signature.
To determine how the alternated lymphocyte distribution could affect metastasis, intensive analysis of T cell subgroup was conducted.
Using this model of tumour metastasis, we examined whether serine protease or deoxyribonuclease I (DNase I) would affect metastasis.
Therefore, we hypothesized that miR-18a might affect tumor growth and metastasis by targeting HIF1A.
The "different cancers into same strain" experiments have allowed cancer biologists to observe how alterations in cancer genes might affect their growth, metabolism, and metastasis.
To test whether NCSTN antibodies might affect the extravasation step of metastasis [ 7], we followed the interaction of MDA-MB-231 cells with endothelial cells by time-lapse microscopy [ 8].
