Sentence examples for middle ear epithelial from inspiring English sources
In this study, we examined the effect of e-liquids on a human middle ear epithelial cell (HMEEC) line.
This study was designed to investigate the anti-inflammatory effect of metformin on lipopolysaccharide (LPS) induced inflammation in human middle ear epithelial cell lines (HMEECs).
To investigate the expression of recently identified human mucin genes in an in vitro model of cultured mouse middle ear epithelial cells (MMEEC).
The aim of this study was to investigate the anti-inflammatory effect of a PPAR-γ agonist, ciglitazone, on cigarette smoke solution (CSS -induced inflammation in human middle ear epithelial CSS -inducedHMEECs).
Although up-regulated expression of S100A8 and S100A9 genes were observed in an animal model of otitis media (OM), their expressions have not been studied in human middle ear epithelial cells in response to the OM pathogen or in patients with recurrent or chronic OM (recurrent OM/RecOM or chronic OM with effusion/COME).
The antibody treated medium was next added to human middle ear epithelial cells transfected with the β-defesnisn 2 promoter construct.
However, DEP have been found to induce TNF- α responses in middle-ear epithelial cells [ 57], but not in human keratinocytes [ 58].
Histological findings indicative of chronic otitis media included frequent remodeling of the middle ear epithelium, characterized by epithelial hyperplasia, polyps and thickening of the middle ear mucosa (Figs 2 and 3).
We recently demonstrated that T phase variants exhibited a higher growth rate and greater epithelial adherence and destruction than did O phase variants during interactions with human middle ear (ME) epithelial cells.
TGIF protein was localized in the nucleus of middle ear cavity epithelial cells in wild-type ears, but as expected not in homozygous mutant ears.
These findings suggest that the expressions of laminin and E-cadherin may be correlated with maturation of the epithelium in the eustachian tube and middle ear and that the epithelial differentiation of the developing murine eustachian tube and middle ear may be controlled by epithelial-mesenchymal interaction and cell-to-cell interaction.
